Pharmacological function of sodium humate
Sodium humate activates the function of the pituitary-adrenal cortex, promotes the secretion of ACTH, increases the secretion of estrogen, and stimulates the balance of the thyroid gland.
- Antibacterial and anti-inflammatory effects. Sodium humate can promote the release of corticosteroids and ACTH by activating the pituitary-adrenal system, inhibiting the synthesis of prostate E (the prostaglandin is a vehicle for painful processes), and is capable of resisting the increase in capillary permeability caused by amines.
- Sodium humate is a polymer complex and contains alkaloids, so it has strong adsorption capacity for intestinal inflammatory substances and toxic substances, and has astringent effect on intestinal mucosa. In addition, since the structure of sodium humate contains various A kind of active group, which can shrink blood vessels, reduce capillary permeability, and play an anti-inflammatory and hemostasis role.
- Under the action of various enzymes, the active group in the sodium humate molecule can decompose more nascent oxygen and exhibit bactericidal action.
- Antiviral effect
Sodium humate, unlike antibiotics, is not decomposed by molds and bacteria, and at the same time denatures viral proteins, thereby producing an antiviral effect.
Sodium humate can prevent DNA viruses (naked and entrapped) from being attached to cells and unable to multiply. In addition, it inhibits reverse transcriptase activity and inhibits virus synthesis.
- Promote wound healing
Sodium humate has a good antiseptic effect on soft-wound suppurative infection. Infected body cells, under the stimulation and damage of microorganisms, local cells lack oxygen to cause necrosis, and isolate harmful ions and poisons. Destroy health and expand the wound. After contact with the wound surface, sodium humate eliminates metal ions and poisons due to changes in physical and chemical properties, and the odor and secretion of pus are also reduced. Sodium humate quickly forms a protective film on the wound surface and shrinks the tiny blood vessels to stop bleeding. Because the molecular structure of sodium humate determines that it is both an oxidant and a reducing agent, it can separate nascent oxygen from the second, increase the internal respiration of cells, promote cell metabolism, and facilitate the survival of inter-ecological cells and the production of granulation. The infection is effectively controlled.
- Sodium humate can increase the number of platelets, shrink blood vessels, reduce capillary permeability, and reverse abnormal microcirculation perfusion to promote normal recovery.
Sodium humate can stimulate the synthesis of prostaglandins in the gastric mucosa, increase the blood flow of the gastric mucosa, protect the gastric mucosa, and heal the ulcer.
- Effect on immune function
Sodium humate can increase the spleen weight of mice and rabbits, enlarge the white pulp area, increase the number of red pulp cells, and produce large macrophages, thereby enhancing the function of immune organs.
- Sodium humate has an excitatory effect on the function of the reticuloendothelial system, ie on non-specific immune function.
- Sodium humate has the effect of enhancing humoral immune function.
- Sodium humate can increase the leaching rate, has a similar effect on phytohemagglutinin (PHA), and participates in cellular immunity.
- Toxicology and detoxification of sodium humate
Sodium humate has been proved to be safe and non-toxic by general toxicity and special toxicity tests. For example, sodium humate is administered orally to rats with LD50? 12g/kg, and humic acid is used to administer LD50? 3150mg/kg in mice. The amount of LD50? 4 g / kg. Sodium humate is not only non-toxic but also detoxifying. It has detoxification effects on heavy metals and strychnine, and can reduce urea toxicity.